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Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes. / Snogdal, Lena Sønder; Wod, Mette; Grarup, Niels; Vestmar, M; Sparsø, Thomas Hempel; Jørgensen, T; Lauritzen, Torsten; Beck-Nielsen, Henning; Henriksen, J E; Pedersen, Oluf; Hansen, Torben; Højlund, K.
In:
Diabetologia, Vol. 55, No. 2, 2012, p. 340-8.
Research output: Contribution to journal › Journal article › Research › peer-review
Harvard
Snogdal, LS, Wod, M
, Grarup, N, Vestmar, M, Sparsø, TH, Jørgensen, T, Lauritzen, T, Beck-Nielsen, H, Henriksen, JE
, Pedersen, O, Hansen, T & Højlund, K 2012, '
Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes',
Diabetologia, vol. 55, no. 2, pp. 340-8.
https://doi.org/10.1007/s00125-011-2377-0APA
Snogdal, L. S., Wod, M.
, Grarup, N., Vestmar, M., Sparsø, T. H., Jørgensen, T., Lauritzen, T., Beck-Nielsen, H., Henriksen, J. E.
, Pedersen, O., Hansen, T., & Højlund, K. (2012).
Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes.
Diabetologia,
55(2), 340-8.
https://doi.org/10.1007/s00125-011-2377-0Vancouver
Snogdal LS, Wod M
, Grarup N, Vestmar M, Sparsø TH, Jørgensen T et al.
Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes.
Diabetologia. 2012;55(2):340-8.
https://doi.org/10.1007/s00125-011-2377-0Author
Snogdal, Lena Sønder ; Wod, Mette ; Grarup, Niels ; Vestmar, M ; Sparsø, Thomas Hempel ; Jørgensen, T ; Lauritzen, Torsten ; Beck-Nielsen, Henning ; Henriksen, J E ; Pedersen, Oluf ; Hansen, Torben ; Højlund, K. / Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes. In: Diabetologia. 2012 ; Vol. 55, No. 2. pp. 340-8.
Bibtex
@article{f9c1ab9571164e6682929502743ef2f3,
title = "Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes",
abstract = "There is substantial evidence that mitochondrial dysfunction is linked to insulin resistance and is present in several tissues relevant to the pathogenesis of type 2 diabetes. Here, we examined whether common variation in genes involved in oxidative phosphorylation (OxPhos) contributes to type 2 diabetes susceptibility or influences diabetes-related metabolic traits.",
author = "Snogdal, {Lena S{\o}nder} and Mette Wod and Niels Grarup and M Vestmar and Spars{\o}, {Thomas Hempel} and T J{\o}rgensen and Torsten Lauritzen and Henning Beck-Nielsen and Henriksen, {J E} and Oluf Pedersen and Torben Hansen and K H{\o}jlund",
year = "2012",
doi = "10.1007/s00125-011-2377-0",
language = "English",
volume = "55",
pages = "340--8",
journal = "Diabetologia",
issn = "0012-186X",
publisher = "Springer",
number = "2",
}
RIS
TY - JOUR
T1 - Common variation in oxidative phosphorylation genes is not a major cause of insulin resistance or type 2 diabetes
AU - Snogdal, Lena Sønder
AU - Wod, Mette
AU - Grarup, Niels
AU - Vestmar, M
AU - Sparsø, Thomas Hempel
AU - Jørgensen, T
AU - Lauritzen, Torsten
AU - Beck-Nielsen, Henning
AU - Henriksen, J E
AU - Pedersen, Oluf
AU - Hansen, Torben
AU - Højlund, K
PY - 2012
Y1 - 2012
N2 - There is substantial evidence that mitochondrial dysfunction is linked to insulin resistance and is present in several tissues relevant to the pathogenesis of type 2 diabetes. Here, we examined whether common variation in genes involved in oxidative phosphorylation (OxPhos) contributes to type 2 diabetes susceptibility or influences diabetes-related metabolic traits.
AB - There is substantial evidence that mitochondrial dysfunction is linked to insulin resistance and is present in several tissues relevant to the pathogenesis of type 2 diabetes. Here, we examined whether common variation in genes involved in oxidative phosphorylation (OxPhos) contributes to type 2 diabetes susceptibility or influences diabetes-related metabolic traits.
U2 - 10.1007/s00125-011-2377-0
DO - 10.1007/s00125-011-2377-0
M3 - Journal article
C2 - 22095239
VL - 55
SP - 340
EP - 348
JO - Diabetologia
JF - Diabetologia
SN - 0012-186X
IS - 2
ER -
