The HETE Is on FFAR1 and Pancreatic Islet Cells
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The HETE Is on FFAR1 and Pancreatic Islet Cells. / Trauelsen, Mette; Lückmann, Michael; Frimurer, Thomas M; Schwartz, Thue W.
In: Cell Metabolism, Vol. 27, No. 2, 2018, p. 273-275.Research output: Contribution to journal › Journal article › Research › peer-review
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TY - JOUR
T1 - The HETE Is on FFAR1 and Pancreatic Islet Cells
AU - Trauelsen, Mette
AU - Lückmann, Michael
AU - Frimurer, Thomas M
AU - Schwartz, Thue W
N1 - Copyright © 2018 Elsevier Inc. All rights reserved.
PY - 2018
Y1 - 2018
N2 - It is known but generally unappreciated that the fatty acid receptor FFAR1 (GPR40) is responsible for a major part of glucose-induced insulin secretion. This puzzling fact is now explained by Tunaru et al. (2018), who demonstrate that glucose-induced 20-hydroxyeicosatetraenoic acid (20-HETE) amplifies insulin secretion through autocrine activation of FFAR1.
AB - It is known but generally unappreciated that the fatty acid receptor FFAR1 (GPR40) is responsible for a major part of glucose-induced insulin secretion. This puzzling fact is now explained by Tunaru et al. (2018), who demonstrate that glucose-induced 20-hydroxyeicosatetraenoic acid (20-HETE) amplifies insulin secretion through autocrine activation of FFAR1.
KW - Journal Article
U2 - 10.1016/j.cmet.2018.01.006
DO - 10.1016/j.cmet.2018.01.006
M3 - Journal article
C2 - 29414682
VL - 27
SP - 273
EP - 275
JO - Cell Metabolism
JF - Cell Metabolism
SN - 1550-4131
IS - 2
ER -
ID: 189764824